IL-17A is a common and critical driver of impaired lung function and immunopathology induced by influenza virus, rhinovirus and respiratory syncytial virus

- Liu, Xiaoming, Nguyen, Thi Hiep, Eyers, Fiona, Foster, Paul S., Yang, Ming, Sokulsky, Leon, Li, Xiang, Netto, Keilah Garcia, Hsu, Alan Chen-Yu, Liu, Chi, Laurie, Karen, Barr, Ian, Tay, Hock

miR-122 promotes virus-induced lung disease by targeting SOCS1

- Collison, Adam M., Sokulsky, Leon A., Johnston, Sebastian L., Foster, Paul S., Mattes, Joerg, Kepreotes, Elizabeth, de Siqueira, Ana Pereira, Morten, Matthew, Edwards, Michael R., Walton, Ross P., Bartlett, Nathan W., Yang, Ming, Nguyen, Thi Hiep

A critical role for the CXCL3/CXCL5/CXCR2 neutrophilic chemotactic axis in the regulation of type 2 responses in a model of rhinoviral-induced asthma exacerbation

- Sokulsky, Leon A, Garcia-Netto, Keilah, Foster, Paul S., Nguyen, Thi Hiep, Girkin, Jason L. N., Collison, Adam, Mattes, Joerg, Kaiko, Gerard, Liu, Chi, Bartlett, Nathan W., Yang, Ming

Identification of IFN-γ and IL-27 as critical regulators of respiratory syncytial virus-induced exacerbation of allergic airways disease in a mouse model

- Nguyen, Thi Hiep, Maltby, Steven, Tay, Hock L., Eyers, Fiona, Foster, Paul S., Yang, Ming

Potential Role of MicroRNAs in the Regulation of Antiviral Responses to influenza infection

- Nguyen, Thi Hiep, Liu, Xiaoming, Su, Zhen Zhong, Hsu, Alan Chen-Yu, Foster, Paul S., Yang, Ming

MicroRNA-21 drives severe, steroid-insensitive experimental asthma by amplifying phosphoinositide 3-kinase-mediated suppression of histone deacetylase 2

- Kim, Richard Y., Horvat, Jay C., Sunkara, Krishna P., Nguyen, Thi Hiep, Jarnicki, Andrew G., Keely, Simon, Mattes, Joerg, Adcock, Ian M., Foster, Paul S., Hansbro, Philip M., Pinkerton, James W., Starkey, Malcolm R., Essilfie, Ama T., Mayall, Jemma R., Nair, Prema M., Hansbro, Nicole G., Jones, Bernadette, Haw, Tatt Jhong

Modeling of respiratory syncytial virus-induced exacerbation of allergic airways disease

- Nguyen, Thi Hiep

Bromodomain and extra terminal (BET) inhibitor suppresses macrophage-driven steroid-resistant exacerbations of airway hyper-responsiveness and inflammation

- Nguyen, Thi Hiep, Maltby, Steven, Eyers, Fiona, Foster, Paul S., Yang, Ming

TNF-α and macrophages are critical for respiratory syncytial virus-induced exacerbations in a mouse model of allergic airways disease

- Nguyen, Thi Hiep, Maltby, Steven, Simpson, Jodie L., Eyers, Fiona, Baines, Katherine J., Gibson, Peter G., Foster, Paul S., Yang, Ming